Hi Dave, Maybe you can help me, and other readers, understand your comments a bit better.
What does "back when I started" mean? Started what?
How many pre-spawning corpses did you actually see in the 70's?
Did the First Nations people used to see a few pre-spawn corpses or 100's?
Do you have a biological degree?
How can a dying liver change the colour of skull cartilage if the liver has not been cut?
How long does it take a dying liver to discolour skull cartilage?
Are you employed in the salmon farming industry?
Maybe Rodney can add his educated opinions ad well please.
Morty, I retired after 37 years with the IPSFC and DFO nearly 4 years ago. Among other duties I provided technical support to DFO, UBC and SFU scientists for research into pre spawning mortality issues relating to Fraser River sockeye. Much of this work involved immune defiencies, stress cortisol measurements, parasite loading and, basic biological cell death.
I do not have a degree; instead, much scientific sampling experience.
I have captured, killed, sampled and histologically examined thousands of adult salmonids over my career.
Salmonid livers and kidneys, along with all other organs, change function in fresh water; with no nutrients (returning adult salmonids cannot digest or metabolize food) to work on the liver retains glycogen and begins to atrophy, become more dense, shrink somewhat and change from the deep blood red color typical of a healthy new -run fish to a green-grey hue, often turning almost yellow. All anglers who have taken early run fish and then, later run fish will have noticed this difference, along with the much larger gall bladder on more sexually mature fish.
Ms. Morton has probably named the condition, “jaundice” correctly. How the liver colors skull tissue I can’t answer; maybe much the way a human with a failing liver would have a yellowish skin color.
This liver breakdown resulting in a yellowish hue is a natural occurrence and is entirely normal. Some fish, especially male pinks, seem to exhibit this.
My statements regarding FN having observed PSM on the Harrison is based on conservations I have had with Chehalis elders. They have long held that Weaver, Big Silver, etc sockeye stocks enter Harrison Lake early and hold below the thermocline, much like Cultus and Chilliwack Lake sockeye. Whenever stocks enter freshwater earlier than normal PSM is common. Death is most often caused by kidney failure due to a parasite named Parvicapsula
No, I do not have any financial interest in BC fish farms but I do support them as I believe they are and will be an important part of BC’s future economy. Wish I could say the same of wild salmonids.